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Experimental Alzheimer's drug shows promise by targeting tau protein

Created at 14 Jul · 1:31 PM1 source↑ Market-relevant
IN SHORT

An experimental drug, diranersen, may slow early Alzheimer's disease by lowering tau protein levels, a different approach than current amyloid-targeting treatments. Early study results suggest it also slowed cognitive decline, prompting plans for larger trials.

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Key Numbers

400people in the diranersen study
26%reduction in cognitive decline with lowest dose

Who's Involved

Biogen
developer of the experimental Alzheimer's drug diranersen
Ionis Pharmaceuticals
partner in the development of diranersen
Jessica Langbaum
Banner Alzheimer's Institute researcher not involved in the study
Dr. Reisa Sperling
Mass General Brigham researcher not involved in the study
Dr. Cath Mummery
University College London researcher who led the new study
University of California, San Francisco
initiator of the Alzheimer's Tau Platform study
Dr. Adam Boxer
UCSF researcher involved in the tau platform study
NewAmsterdam Pharma
maker of the experimental drug obicetrapib
Denali Therapeutics
pursuing drugs using a 'transport vehicle' technology

↳ Why This Matters

The development of diranersen offers a new potential avenue for treating Alzheimer's disease, addressing the tau protein which is implicated in the disease's progression. This could lead to more effective therapies, especially for patients who do not respond well to or cannot tolerate existing amyloid-targeting drugs.

Key facts

  • An experimental drug, diranersen, targets tau protein levels in the brain to potentially slow early Alzheimer's disease.
  • Unlike current treatments focusing on amyloid, diranersen aims to reduce the production of abnormal tau.
  • Early study results indicated diranersen slowed cognitive decline in some participants, with one dose showing a 26% reduction.
  • The drug is administered via injection into the fluid surrounding the spinal cord.
  • Side effects were noted, including injection site pain and temporary confusion, but no brain inflammation.
  • New research initiatives, including a tau vaccine and other novel approaches, are also being explored.

An experimental drug called diranersen is showing promise in potentially slowing the progression of early Alzheimer's disease by targeting the tau protein, a different mechanism than current treatments that focus on amyloid buildup. Researchers presented findings suggesting that diranersen not only lowers tau levels but also modestly slows cognitive decline, with one small subset of participants showing a reduction comparable to amyloid therapies.

The study, involving approximately 400 individuals with mild cognitive impairment or mild Alzheimer's, found that five out of six cognitive tests indicated slower decline in recipients compared to placebo. Notably, the lowest dose, administered every six months, demonstrated a 26% reduction in cognitive decline, a result considered encouraging despite the study not meeting its primary goal of dose-dependent benefits.

Diranersen, an antisense oligonucleotide, works by instructing a tau-producing gene to generate less of the protein. This approach differs from current infusions or injections of anti-amyloid drugs, as diranersen is injected into the spinal fluid for a more direct path to the brain. Side effects were reported, including injection site pain and temporary confusion, but importantly, no signs of brain inflammation were observed, unlike those sometimes seen with anti-amyloid treatments.

This development is spurring renewed interest in tau-targeting mechanisms, with other novel approaches also under investigation. These include a tau vaccine being tested in a new platform study by the University of California, San Francisco, and an experimental cholesterol-lowering drug, obicetrapib, being explored for its potential to mitigate Alzheimer's risk in individuals with a genetic predisposition.

Companies are also exploring innovative drug delivery methods, such as Denali Therapeutics' approach to use natural transport mechanisms, like iron, to ferry drugs across the blood-brain barrier more effectively.

Frequently asked questions

Diranersen is an experimental drug, an antisense oligonucleotide, designed to slow early Alzheimer's disease by instructing a tau-producing gene to produce less tau protein.

Current Alzheimer's drugs like lecanemab and donanemab target amyloid protein buildup, while diranersen targets tau protein levels. Diranersen is also injected into spinal fluid, unlike the bloodstream administration of some amyloid drugs.

The study found that diranersen recipients' memory and cognitive abilities worsened more slowly than those given placebo shots. One low dose showed a 26% reduction in cognitive decline.

Side effects included injection site pain and a temporary state of confusion that could appear a few days after the shot. There were no signs of brain inflammation.

What Happens Next

01Biogen plans to conduct a larger study to further prove diranersen's benefit.
02The Alzheimer's Tau Platform study will expand to include more locations and other tau drugs.
03NewAmsterdam Pharma plans to begin a study testing obicetrapib's effect on Alzheimer's risk in specific genetic groups.

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Cadence

How It Developed

Researchers reported an experimental drug, diranersen, may slow early Alzheimer's by lowering tau protein levels.
The drug, diranersen, is an antisense oligonucleotide that instructs a tau-producing gene to produce less tau.
A study of about 400 people found diranersen also slowed cognitive decline in a subset of participants.
In one test of the lowest dose, diranersen showed a 26% reduction in cognitive decline.
Side effects included injection site pain and temporary confusion, but no signs of brain inflammation.
The University of California, San Francisco, opened a study to test various experimental anti-tau therapies.
An experimental cholesterol-lowering drug, obicetrapib, is being explored for its potential to lower Alzheimer's-related proteins.

Sources

T1
An experimental Alzheimer’s drug shows some promise as researchers hunt new approachesAP News

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